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  • Publisher Correction
  • Open Access

Publisher Correction: Is DNA methylation the new guardian of the genome?

Molecular Cytogenetics201811:38

https://doi.org/10.1186/s13039-018-0385-1

  • Received: 21 May 2018
  • Accepted: 22 May 2018
  • Published:

The original article was published in Molecular Cytogenetics 2017 10:11

Correction

In the original publication of this article [1] the figures and the captions of 3 figures do not match correctly due to a typographical error. In this correction article the corrected figures and captions for Figs. 1, 2 and 3 are shown.
Fig. 1
Fig. 1

Rates of transmethylation of human tumor cell lines and normal human fibroblast cell strains. All cells were labeled with 100 μM [35S]-methionine-containing medium (25 μCi/ml) for 24 h. Periodateoxidized 3-deazaadenosine was added to a concentration of 10 µM and the accumulation of [35S] AdoHcy was measured at half- hour intervals. Solid lines are human cancer cell lines. Dashed lines are human normal cell strains [38]

Fig. 2
Fig. 2

Recombinant methioninase (rMETase) traps cancer cells in S/G2 phase. Time-course imaging of HeLa-FUCCI cells treated with rMETase (1.0 unit/ml). Kinetics of rMETase trapping of cells in S/G2. Images were acquired with the FV1000 confocal microscope (Olympus, Tokyo, Japan). In the FUCCI system, the cells in G0/G1, S, or G2/M phases appear red, yellow, or green, respectively [66]

Fig. 3
Fig. 3

Efficacy of recombinant methioninase (rMETase) on growth of human colon tumors HCT 15 in nude mice. rMETase (5 or 10 units/g every 8 h) was administered by i.p. injection in nude mice with human colon tumor HCT 15, growing s.c. [54]

The publisher apologizes to the readers and authors for the inconvenience.

Notes

Declarations

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Authors’ Affiliations

(1)
AntiCancer Inc., 7917 Ostrow Street, San Diego, CA 92111, USA
(2)
Department of Surgery, University of California, San Diego, CA, USA

Reference

  1. Hoffman RM. Mol Cytogenet. 2017;10(11) https://doi.org/10.1186/s13039-017-0314-8.

Copyright

© The Author(s). 2018

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